Table of contents

1. Introduction to Microbiology3h 22m
2. Disproving Spontaneous Generation1h 18m
3. Chemical Principles of Microbiology3h 38m
4. Water1h 28m
5. Molecules of Microbiology2h 23m
6. Cell Membrane & Transport3h 28m
7. Prokaryotic Cell Structures & Functions5h 52m
8. Eukaryotic Cell Structures & Functions2h 18m
9. Microscopes2h 46m
10. Dynamics of Microbial Growth4h 36m
11. Controlling Microbial Growth4h 10m
12. Microbial Metabolism5h 16m
13. Photosynthesis2h 31m
14. DNA Replication2h 25m
15. Central Dogma & Gene Regulation7h 14m
16. Microbial Genetics4h 44m
17. Biotechnology3h 0m
18. Viruses, Viroids, & Prions4h 56m
19. Innate Immunity7h 15m
20. Adaptive Immunity7h 14m
21. Principles of Disease6h 57m

19. Innate Immunity

Phagoctytosis

Microbiology

19. Innate Immunity

Phagoctytosis

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Multiple Choice

Some pathogens create C5a peptidase, an enzyme that destroys C5a proteins. Which of the following is not a benefit that the pathogen would experience after destroying C5a proteins?

Without C5a, the pathogen is less likely to attract a phagocytotic cell via chemotaxis.

Without C5a, complement proteins and immune cells are less likely to come to the site of infection.

C5a would not be able to bind to the pathogen and trigger opsonization.

Without C5a, mast cells are less likely to release histamines & cytokines which trigger inflammation.

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Verified step by step guidance

Understand the role of C5a in the immune response. C5a is a potent chemoattractant that recruits immune cells to the site of infection and enhances inflammation.

Recognize that C5a peptidase is an enzyme produced by some pathogens to degrade C5a, thereby evading the immune response.

Evaluate each statement to determine how the absence of C5a affects the immune response: (1) Less attraction of phagocytic cells, (2) Reduced recruitment of complement proteins and immune cells, (3) Inability to bind and trigger opsonization, (4) Decreased release of histamines and cytokines.

Identify that C5a primarily functions in chemotaxis and inflammation, not directly in opsonization. Opsonization involves marking pathogens for phagocytosis, typically through other complement components like C3b.

Conclude that the statement about C5a not being able to bind and trigger opsonization is incorrect because C5a's main role is not in opsonization, but in recruiting immune cells and promoting inflammation.