Table of contents

1. Introduction to Microbiology3h 22m
2. Disproving Spontaneous Generation1h 18m
3. Chemical Principles of Microbiology3h 38m
4. Water1h 28m
5. Molecules of Microbiology2h 23m
6. Cell Membrane & Transport3h 28m
7. Prokaryotic Cell Structures & Functions5h 52m
8. Eukaryotic Cell Structures & Functions2h 18m
9. Microscopes2h 46m
10. Dynamics of Microbial Growth4h 36m
11. Controlling Microbial Growth4h 10m
12. Microbial Metabolism5h 16m
13. Photosynthesis2h 31m
14. DNA Replication2h 25m
15. Central Dogma & Gene Regulation7h 14m
16. Microbial Genetics4h 44m
17. Biotechnology3h 0m
18. Viruses, Viroids, & Prions4h 56m
19. Innate Immunity7h 15m
20. Adaptive Immunity7h 14m
21. Principles of Disease6h 57m

21. Principles of Disease

4) Avoiding Complement System

Microbiology

21. Principles of Disease

4) Avoiding Complement System

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Multiple Choice

All of these survival mechanisms are unique to serum resistant bacterial pathogens except which of the following?

Producing membrane damaging toxins.

Bacterial proteins binding to C3b.

Thick peptidoglycan cells walls.

Inhibition of C3 convertase.

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Verified step by step guidance

Understand the context: Serum resistance in bacteria refers to the ability of certain bacteria to survive and proliferate in the presence of host serum, which contains immune components like complement proteins.

Identify the role of complement system: The complement system is a part of the immune system that enhances the ability to clear microbes and damaged cells. It involves proteins like C3b and C3 convertase, which play crucial roles in opsonization and the formation of the membrane attack complex.

Analyze each option: Consider how each mechanism contributes to serum resistance. For example, bacterial proteins binding to C3b can prevent opsonization, thick peptidoglycan cell walls can provide physical protection, and inhibition of C3 convertase can prevent the formation of the membrane attack complex.

Evaluate the exception: Producing membrane-damaging toxins is a mechanism that bacteria use to damage host cells, but it is not specifically a mechanism for resisting serum or complement-mediated killing.

Conclude which mechanism is not unique to serum resistance: Based on the analysis, identify that producing membrane-damaging toxins is not a unique mechanism for serum resistance, as it is more related to direct host cell damage rather than evading the complement system.