In a population of 2500, how many babies would you expect to have cystic fibrosis, a homozygous recessive condition, if the frequency of the dominant allele is 0.9 and the population is at Hardy–Weinberg equilibrium?a. 0.9×2500=2250b. 2×0.9×0.1×2500=450c. 0.9×0.1×2500=225d. 0.1x0.1x2500=25

If the nucleotide variability of a locus equals 0%, what is the gene variability and number of alleles at that locus?a. genevariability=0%;numberofalleles=0b. genevariability=0%;numberofalleles=1c. genevariability=0%;numberofalleles=2d. genevariability>0%;numberofalleles=2

There are 25 individuals in population 1, all with genotype AA, and there are 40 individuals in population 2, all with genotype aa. Assume that these populations are located far from each other and that their environmental conditions are very similar. Based on the information given here, the observed genetic variation most likely resulted froma. genetic drift.b. gene flow.c.nonrandom mating.d. directional selection.

Single strands of nucleic acids are directional, meaning that there are two different ends. What functional groups define the two different ends of a strand?

A fruit fly population has a gene with two alleles, A1 and A2. Tests show that 70% of the gametes produced in the population contain the A1 allele. If the population is in Hardy-Weinberg equilibrium, what proportion of the flies carry both A1 and A2?a. 0.7b. 0.49c. 0.42d. 0.21

In a population with two alleles, B and b, the allele frequency of b is 0.4. B is dominant to b. What is the frequency of individuals with the dominant phenotype if the population is in Hardy-Weinberg equilibrium?a. 0.16b. 0.36c. 0.48d. 0.84

Determine what is incorrect in the following statement: Red deer mutated their genes so that they could have larger antlers to compete for mates.

Make a concept map (see BioSkills 12) that relates DNA's primary structure to its secondary structure. Your diagram should include deoxyribonucleotides, base-stacking interactions, purines, pyrimidines, phosphodiester linkages, DNA's primary structure, DNA's secondary structure, complementary base pairing, and antiparallel double helix.

In humans, albinism is caused by loss-of-function mutations in genes involved in the synthesis of melanin, the dark pigment in skin. Only people homozygous for a loss-of-function allele (genotype aa) have the albino phenotype. In Americans of northern European ancestry, albino individuals are present at a frequency of about 1 in 10,000 (or 0.0001). Assuming that genotypes are in Hardy–Weinberg equilibrium, what is the predicted frequency of Caucasians in the United States who carry a single allele for albinism?